Icariin
January 16, 2020Gossypol
January 17, 2020
- Exercise prevents diet-induced cellular senescence in adipose tissue
- Physiological aging: links among adipose tissue dysfunction, diabetes, and frailty
- Unbiased analysis of senescence associated secretory phenotype (SASP) to identify common components following different genotoxic stresses
- Aging and adipose tissue: potential interventions for diabetes and regenerative medicine
- Roles of chemokine ligand-2 (CXCL2) and neutrophils in influencing endothelial cell function and inflammation of human adipose tissue
- Ablation of XP-V gene causes adipose tissue senescence and metabolic abnormalities
- The emerging role of senescent cells in tissue homeostasis and pathophysiology
- Cellular senescence and the senescent secretory phenotype in age-related chronic diseases
- JAK inhibition alleviates the cellular senescence-associated secretory phenotype and frailty in old age
- Growth hormone action predicts age-related white adipose tissue dysfunction and senescent cell burden in mice
- Aging-related inflammation in osteoarthritis
- Cellular senescence in type 2 diabetes: a therapeutic opportunity
- The fat cell senescence hypothesis: a mechanism responsible for abrogating the resolution of inflammation in chronic disease
- Feast and famine: adipose tissue adaptations for healthy aging
- Macrophages in age-related chronic inflammatory diseases
- Proposed metabolic vicious circle in patients with large myocardial infarcts and high plasma-free-fatty-acid concentrations